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Jimmy J Lin, 451819 Panarama Ct, Mclean, VA 22101

Jimmy Lin Phones & Addresses

McLean, VA   

Silver Spring, MD   

Cambridge, MA   

Bethesda, MD   

Fairfax, VA   

Boston, MA   

Mentions for Jimmy J Lin

Career records & work history

Medicine Doctors

Jimmy J. Lin

Specialties:
Internal Medicine
Work:
Jimmy J Lin MD
2411 Frst Ave, San Jose, CA 95128
408-9831012 (phone) 408-9831021 (fax)
Education:
Medical School
University of the East - Ramon Magsaysay Memorial Medical Center, Quezon City, Philippines
Graduated: 1980
Procedures:
Continuous EKG, Electrocardiogram (EKG or ECG), Vaccine Administration
Conditions:
Acne, Acute Bronchitis, Acute Sinusitis, Atrial Fibrillation and Atrial Flutter, Benign Prostatic Hypertrophy, Calculus of the Urinary System, Chronic Bronchitis, Chronic Renal Disease, Contact Dermatitis, Depressive Disorders, Diabetes Mellitus (DM), Disorders of Lipoid Metabolism, Fractures, Dislocations, Derangement, and Sprains, Gastritis and Duodenitis, Gastroesophageal Reflux Disease (GERD), Gout, Hemorrhoids, Herpes Zoster, Hypertension (HTN), Hypothyroidism, Infectious Liver Disease, Ischemic Heart Disease, Labyrinthitis, Osteoporosis, Vitamin D Deficiency, Abdominal Hernia, Abnormal Vaginal Bleeding, Acute Conjunctivitis, Acute Glomerulonephritis, Acute Pharyngitis, Acute Upper Respiratory Tract Infections, Alopecia Areata, Alzheimer's Disease, Anal or Rectal Abscess, Angina Pectoris, Anxiety Dissociative and Somatoform Disorders, Anxiety Phobic Disorders, Arterial Thromboembolic Disease, Atherosclerosis, Atopic Dermatitis, Bell's Palsy, Benign Paroxysmal Positional Vertigo, Bipolar Disorder, Bronchial Asthma, Candidiasis, Candidiasis of Vulva and Vagina, Cardiac Arrhythmia, Carpel Tunnel Syndrome, Cataract, Cholelethiasis or Cholecystitis, Chronic Fatigue Syndrome, Cirrhosis, Conduction Disorders, Constipation, Dementia, Dermatitis, Diabetic Peripheral Neuropathy, Diverticulitis, Diverticulosis, Erectile Dysfunction (ED), Gastrointestinal Hemorrhage, Gingival and Periodontal Diseases, Glaucoma, Hearing Loss, Heart Failure, Hemorrhagic stroke, Herpes Simplex, HIV Infection, Hyperthyroidism, Inflammatory Bowel Disease (IBD), Influenza, Inguinal Hernia, Insomnia, Intervertebral Disc Degeneration, Iron Deficiency Anemia, Irritable Bowel Syndrome (IBS), Keratitis, Malignant Neoplasm of Female Breast, Menopausal and Postmenopausal Disorders, Metabolic Syndrome, Migraine Headache, Non-Toxic Goiter, Osteoarthritis, Otitis Media, Overweight and Obesity, Parkinson's Disease, Paroxysmal Supreventricular Tachycardia (PSVT), Peripheral Nerve Disorders, Phlebitis and Thrombophlebitis, Plantar Warts, Pneumonia, Prostatitis, Psoriasis, Pulmonary Embolism, Pulmonary Tuberculosis (TB), Rheumatoid Arthritis, Rotator Cuff Syndrome and Allied Disorders, Sciatica, Septicemia, Skin and Subcutaneous Infections, Skin Cancer, Systemic Lupus Erythematosus, Tension Headache, Tinea Pedis, Transient Cerebral Ischemia, Urinary Incontinence, Varicose Veins, Venous Embolism and Thrombosis, Ventral Hernia, Viral Meningitis, Vitamin B12 Deficiency Anemia
Languages:
Chinese, English
Description:
Dr. Lin graduated from the University of the East - Ramon Magsaysay Memorial Medical Center, Quezon City, Philippines in 1980. He works in San Jose, CA and specializes in Internal Medicine. Dr. Lin is affiliated with OConnor Hospital.

Jimmy Lin resumes & CV records

Resumes

Jimmy Lin Photo 46

Management Consultant At Zs Associates

Location:
Washington D.C. Metro Area
Industry:
Management Consulting
Jimmy Lin Photo 47

Chief Technologist

Location:
Washington, DC
Industry:
Information Technology And Services
Work:
Booz Allen Hamilton since Jul 2006
Associate
United InnoWorks Academy Jan 2007 - Dec 2010
Chief Information Officer
Harris Corporation Mar 2004 - Dec 2005
Senior Systems Analyst
Education:
University of Maryland College Park 1998 - 2003
Bachelors, Mechanical Engineering
Skills:
Active Dod Top Secret Clearance, Enterprise Architecture, Project Management, Cloud Computing, Big Data, Web Development, Css, Twitter Bootstrap, Javascript, Node.js, Jquery, Python, Mysql, Java, Git, Puppet, Hadoop, Acummulo, Solr, Elasticsearch, Apache Spark, Regular Expressions, Rest, Linux, User Experience, Agile Project Management, Mongodb, Amazon Web Services, Alexa, Docker, Devops, Jenkins
Interests:
Volleyball
League of Legends
Web Development
Education
Environment
Starcraft
Science and Technology
Stackoverflow
Washington Redskins
Data Management
Slashdot
Languages:
Mandarin
Certifications:
Aws Certified Developer - Associate
Amazon Web Services
Jimmy Lin Photo 48

President

Industry:
Food Production
Work:
Lin's Hawaiian Snacks
President
Jimmy Lin Photo 49

Jimmy Lin

Jimmy Lin Photo 50

Jimmy Lin

Jimmy Lin Photo 51

Jimmy Lin

Jimmy Lin Photo 52

Jimmy Lin

Location:
Seattle, Washington
Industry:
Marketing and Advertising
Jimmy Lin Photo 53

Jimmy Lin

Location:
United States

Publications & IP owners

Wikipedia

Jimmy Lin Photo 59

Christopher Lee (Singaporean Actor)

… Male Artiste at the Star Awards 1998 for his performance. Since then, Lee has acted in many television series jointly-produced by Singapore and other countries in the Asia-Pacific region, co-starring with actors from Hong Kong and Taiwan, such as Nadia Chan, Vincent Jiao, Jimmy Lin and Jord...

Us Patents

Genomic Landscapes Of Human Breast And Colorectal Cancers

US Patent:
2009012, May 14, 2009
Filed:
Oct 8, 2008
Appl. No.:
12/247464
Inventors:
Laura D. Wood - Baltimore MD, US
Williams D. Parsons - Ellicott City MD, US
Sian Jones - Baltimore MD, US
Jimmy Lin - Baltimore MD, US
Tobias Sjoblom - Uppsala, SE
Thomas Barber - Noblesville IN, US
Giovanni Parmigiani - Baltimore MD, US
Victor Velculescu - Dayton MD, US
Kenneth W. Kinzler - Bel Air MD, US
Bert Vogelstein - Baltimore MD, US
Assignee:
The Johns Hopkins University - Baltimore MD
International Classification:
C12Q 1/68
C12Q 1/02
US Classification:
435 6, 435 29
Abstract:
Human cancer is caused by the accumulation of mutations in oncogenes and tumor suppressor genes. To catalogue the genetic changes that occur during tumorigenesis, we isolated DNA from 11 breast and 11 colorectal tumors and determined the sequences of the genes in the Reference Sequence database in these samples. Based on analysis of exons representing 20,857 transcripts from 18,191 genes, we conclude that the genomic landscapes of breast and colorectal cancers are composed of a handful of commonly mutated gene “mountains” and a much larger number of gene “hills” that are mutated at low frequency. We describe statistical and bioinformatic tools that may help identify mutations with a role in tumorigenesis. These results have implications for understanding the nature and heterogeneity of human cancers and for using personal genomics for tumor diagnosis and therapy.

Consensus Coding Sequences Of Human Breast And Colorectal Cancers

US Patent:
2010031, Dec 16, 2010
Filed:
Aug 13, 2007
Appl. No.:
12/377073
Inventors:
Tobias Sjoblom - Baltimore MD, US
Sian Jones - Baltimore MD, US
D. Williams Parsons - Baltimore MD, US
Laura D. Wood - Baltimore MD, US
Jimmy Lin - Baltimore MD, US
Thomas Barber - Baltimore MD, US
Diana Mandelker - Baltimore MD, US
Bert Vogelstein - Baltimore MD, US
Kenneth W. Kinzler - Baltimore MD, US
Assignee:
JOHNS HOPKINS UNIVERSITY - Baltimore MD
International Classification:
C12Q 1/68
US Classification:
435 6
Abstract:
Analysis of 13,023 genes in 11 breast and 11 colorectal cancers revealed that individual tumors accumulate an average of 90 mutant genes but that only a subset of these contribute to the neoplastic process. Using stringent criteria to delineate this subset, we identified 189 genes (average of 11 per tumor) that were mutated at significant frequency. The vast majority of these genes were not known to be genetically altered in tumors and are predicted to affect a wide range of cellular functions, including transcription, adhesion, and invasion. These data define the genetic landscape of two human cancer types, provide new targets for diagnostic and therapeutic intervention and monitoring.

Genetic Alterations In Isocitrate Dehydrogenase And Other Genes In Malignant Glioma

US Patent:
2011022, Sep 22, 2011
Filed:
Sep 3, 2009
Appl. No.:
13/060191
Inventors:
Bert Vogelstein - Baltimore MD, US
Kenneth W. Kinzler - Baltimore MD, US
D. Williams Parsons - Ellicott City MD, US
Xiaosong Zhang - Baltimore MD, US
Jimmy Cheng-Ho Lin - Baltimore MD, US
Rebecca J. Leary - Baltimore MD, US
Philipp Angenendt - Baltimore MD, US
Nickolas Papadopoulos - Towson MD, US
Victor Velculescu - Dayton MD, US
Giovanni Parmigiani - Baltimore MD, US
Rachel Karchin - Towson MD, US
Sian Jones - Baltimore MD, US
Hai Yan - Chapel Hill NC, US
Darell Bigner - Mebane NC, US
Chien-Tsun Kuan - Cary NC, US
Assignee:
THE JOHNS HOPKINS UNIVERSITY - Baltimore MD
DUKE UNIVERSITY - Durham SC
International Classification:
A61K 39/395
C12Q 1/68
G01N 33/574
C07K 16/32
A61K 39/00
C12N 9/04
C07H 21/04
C12Q 1/32
A61P 37/04
A61P 35/00
US Classification:
4241381, 435 618, 4241741, 435 71, 435 611, 5303877, 4241851, 435190, 536 232, 435 26
Abstract:
We found mutations of the R132 residue of isocitrate dehydrogenase 1 (IDH1) in the majority of grade II and III astrocytomas and oligodendrogliomas as well as in glioblastomas that develop from these lower grade lesions. Those tumors without mutations in IDH1 often had mutations at the analogous R172 residue of the closely related IDH2 gene. These findings have important implications for the pathogenesis and diagnosis of malignant gliomas.

Pathways Underlying Pancreatic Tumorigenesis And An Hereditary Pancreatic Cancer Gene

US Patent:
2012011, May 10, 2012
Filed:
Sep 3, 2009
Appl. No.:
13/060453
Inventors:
Bert Vogelstein - Baltimore MD, US
Kenneth W. Kinzler - Baltimore MD, US
D. Williams Parsons - Ellicott City MD, US
Sian Jones - Baltimore MD, US
Xiaosong Zhang - Baltimore MD, US
Jimmy Cheng-Ho Lin - Baltimore MD, US
Rebecca J. Leary - Baltimore MD, US
Philipp Angenendt - Hamburg, DE
Nickolas Papadopoulos - Towson MD, US
Victor Velculescu - Dayton MD, US
Giovanni Parmigiani - Brookline MA, US
Rachel Karchin - Towson MD, US
Scott Kern - Hunt Valley MD, US
Ralph Hruban - Baltimore MD, US
James R. Eshleman - Lutherville MD, US
Michael Goggins - Baltimore MD, US
Alison Klein - Baltimore MD, US
Assignee:
THE JOHNS HOPKINS UNIVERSITY - Baltimore MD
International Classification:
C12Q 1/68
C40B 20/00
C07H 21/04
US Classification:
506 2, 435 611, 536 2431, 536 2433
Abstract:
There are currently few therapeutic options for patients with pancreatic cancers and new insights into the pathogenesis of this lethal disease are urgently needed. To this end, we performed a comprehensive analysis of the genes altered in 24 pancreatic tumors. First, we determined the sequences of 23,781 transcripts, representing 20,583 protein-encoding genes, in DNA from these tumors. Second, we searched for homozygous deletions and amplifications using microarrays querying one million single nucleotide polymorphisms in each sample. Third, we analyzed the transcriptomes of the same samples using SAGE and next-generation sequencing-by-synthesis technologies. We found that pancreatic cancers contain an average of 63 genetic alterations, of which 49 are point mutations, 8 are homozygous deletions, and 6 are amplifications. Further analyses revealed a core set of 12 regulatory processes or pathways that were each genetically altered in 70% to 100% of the samples. The data suggest that dysregulation of this core set of pathways is responsible for the major features of pancreatic tumorigenesis.

Genetic Alterations In Isocitrate Dehydrogenase And Other Genes In Malignant Glioma

US Patent:
2012020, Aug 9, 2012
Filed:
Mar 6, 2012
Appl. No.:
13/412696
Inventors:
Bert VOGELSTEIN - Baltimore MD, US
Kenneth W. KINZLER - Baltimore MD, US
D. Williams PARSONS - Bellaire TX, US
Xiaosong ZHANG - Baltimore MD, US
Jimmy Cheng-Ho LIN - Baltimore MD, US
Rebecca J. LEARY - Baltimore MD, US
Philipp ANGENENDT - Hamburg, DE
Nickolas PAPADOPOULOS - Towson MD, US
Victor VELCULESCU - Dayton MD, US
Giovanni PARMIGIANI - Boston MA, US
Rachel KARCHIN - Towson MD, US
Sian JONES - Baltimore MD, US
Hai YAN - Chapel Hill NC, US
Darell BIGNER - Mebane NC, US
Chien-Tsun KUAN - Cary NC, US
Gregory J. RIGGINS - White Hall MD, US
Assignee:
DUKE UNIVERSITY - Durham SC
THE JOHNS HOPKINS UNIVERSITY - Baltimore MD
International Classification:
C12Q 1/68
C07K 16/40
C07H 21/04
G01N 33/574
US Classification:
435 611, 435 612, 435 74, 5303898, 53038826, 5303873, 536 232
Abstract:
We found mutations of the R132 residue of isocitrate dehydrogenase 1 (IDH1) in the majority of grade II and III astrocytomas and oligodendrogliomas as well as in glioblastomas that develop from these lower grade lesions. Those tumors without mutations in IDH1 often had mutations at the analogous R172 residue of the closely related IDH2 gene. These findings have important implications for the pathogenesis and diagnosis of malignant gliomas.

Genomic Landscapes Of Human Breast And Colorectal Cancers

US Patent:
2013019, Aug 1, 2013
Filed:
Sep 28, 2011
Appl. No.:
13/247552
Inventors:
Laura D. WOOD - Baltimore MD, US
Williams D. PARSONS - Ellicott City MD, US
Sian JONES - Baltimore MD, US
Jimmy LIN - Baltimore MD, US
Tobias SJÖBLOM - Uppsala, SE
Thomas BARBER - Noblesville IN, US
Giovanni PARMIGIANI - Baltimore MD, US
Victor VELCULESCU - Dayton MD, US
Kenneth W. KINZLER - Bel Air MD, US
Bert VOGELSTEIN - Baltimore MD, US
Assignee:
THE JOHNS HOPKINS UNIVERSITY - Baltimore MD
International Classification:
C12Q 1/68
US Classification:
435 611
Abstract:
Human cancer is caused by the accumulation of mutations in oncogenes and tumor suppressor genes. To catalogue the genetic changes that occur during tumorigenesis, we isolated DNA from 11 breast and 11 colorectal tumors and determined the sequences of the genes in the Reference Sequence database in these samples. Based on analysis of exons representing 20,857 transcripts from 18,191 genes, we conclude that the genomic landscapes of breast and colorectal cancers are composed of a handful of commonly mutated gene “mountains” and a much larger number of gene “hills” that are mutated at low frequency. We describe statistical and bioinformatic tools that may help identify mutations with a role in tumorigenesis. These results have implications for understanding the nature and heterogeneity of human cancers and for using personal genomics for tumor diagnosis and therapy.

Consensus Coding Sequences Of Human Breast And Colorectal Cancers

US Patent:
2020023, Jul 30, 2020
Filed:
Mar 19, 2020
Appl. No.:
16/824052
Inventors:
- Baltimore MD, US
Sian JONES - Baltimore MD, US
D. Williams PARSONS - Bellaire TX, US
Laura D. WOOD - Baltimore MD, US
Jimmy Cheng-Ho LIN - Baltimore MD, US
Thomas BARBER - Nobelsville TN, US
Diana MANDELKER - Baltimore MD, US
Bert VOGELSTEIN - Baltimore MD, US
Kenneth W. KINZLER - Baltimore MD, US
Victor E. VELCULESCU - Dayton MD, US
International Classification:
C12Q 1/6886
Abstract:
Analysis of 13,023 genes in 11 breast and 11 colorectal cancers revealed that individual tumors accumulate an average of 90 mutant genes but that only a subset of these contribute to the neoplastic process. Using stringent criteria to delineate this subset, we identified 189 genes (average of 11 per tumor) that were mutated at significant frequency. The vast majority of these genes were not known to be genetically altered in tumors and are predicted to affect a wide range of cellular functions, including transcription, adhesion, and invasion. These data define the genetic landscape of two human cancer types, provide new targets for diagnostic and therapeutic intervention and monitoring.

Consensus Coding Sequences Of Human Breast And Colorectal Cancers

US Patent:
2020004, Feb 13, 2020
Filed:
Oct 25, 2019
Appl. No.:
16/664505
Inventors:
- Baltimore MD, US
Sian JONES - Baltimore MD, US
D. Williams PARSONS - Bellaire TX, US
Laura D. WOOD - Baltimore MD, US
Jimmy Cheng-Ho LIN - Baltimore MD, US
Thomas BARBER - Nobelsville TN, US
Diana MANDELKER - Baltimore MD, US
Bert VOGELSTEIN - Baltimore MD, US
Kenneth W. KINZLER - Baltimore MD, US
Victor E. VELCULESCU - Dayton MD, US
International Classification:
C12Q 1/6886
Abstract:
Analysis of 13,023 genes in 11 breast and 11 colorectal cancers revealed that individual tumors accumulate an average of 90 mutant genes but that only a subset of these contribute to the neoplastic process. Using stringent criteria to delineate this subset, we identified 189 genes (average of 11 per tumor) that were mutated at significant frequency. The vast majority of these genes were not known to be genetically altered in tumors and are predicted to affect a wide range of cellular functions, including transcription, adhesion, and invasion. These data define the genetic landscape of two human cancer types, provide new targets for diagnostic and therapeutic intervention and monitoring.

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