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Yu F Li, 676707 Homestake Dr, Bowie, MD 20720

Yu Li Phones & Addresses

Crofton, MD   

Bowie, MD   

Rockville, MD   

Gaithersburg, MD   

Clarksville, MD   

Ellicott City, MD   

Laurel, MD   

Mentions for Yu F Li

Career records & work history

Lawyers & Attorneys

Yu Li Photo 1

Yu Li, Washington DC - Lawyer

Address:
Curtis Mallet-Prevost, Colt & Mosle LLP
1717 Pennsylvania Ave Nw, Washington, DC 20006
202-4527355 (Office)
Licenses:
New York - Currently registered 2005
Education:
University of Wisconsin Law School
Specialties:
Advertising - 34%
Commercial - 33%
Government - 33%
Yu Li Photo 2

Yu Li - Lawyer

Address:
391-1527651 (Office)
Licenses:
Wisconsin - Good Standing 2004
Education:
University of Wisconsin Law SchoolGraduated 2004
Languages:
English, Mandarin
Yu Li Photo 3

Yu Li - Lawyer

Licenses:
New York - Currently registered 2013
Education:
Indiana University School of Law

Medicine Doctors

Yu J. Li

Description:
Dr. Li graduated from the China Med Univ, Shenyang City, Liaoning, China in 1985. He works in Davenport, IA and 1 other location and specializes in Anatomic Pathology & Clinical Pathology. Dr. Li is affiliated with Trinity Medical Center Moline, Unitypoint Health Trinity, UnityPoint Health Trinity Bettendorf and Unitypoint Health Trinity Regional Medical Center.

Publications & IP owners

Us Patents

Function Of Autophagy Genes In Cell Death

US Patent:
7838645, Nov 23, 2010
Filed:
May 2, 2005
Appl. No.:
11/119569
Inventors:
Eric H. Baehrecke - University Park MD, US
Ajjai Alva - Baltimore MD, US
Michael J. Lenardo - Potomac MD, US
Yu Li - Rockville MD, US
Assignee:
University of Maryland College Park - College Park MD
National Institutes of Health - Bethesda MD
International Classification:
C07H 21/02
C07K 16/00
US Classification:
536 231, 5303871
Abstract:
The present invention relates to a new molecular pathway in which activation of the receptor-interacting protein (RIP, a serine-threonine kinase) and Jun N-terminal kinase induce cell death with the morphology of autophagy. Further, autophagic death is induced by caspase 8 inhibition and expression of the mammalian genes ATG7 and beclin.

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